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transmission 268 electron microscope  (Hitachi Ltd)


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  • 99

    Structured Review

    Hitachi Ltd transmission 268 electron microscope
    Transmission 268 Electron Microscope, supplied by Hitachi Ltd, used in various techniques. Bioz Stars score: 99/100, based on 48653 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/transmission+268+electron+microscope/HT7800/pm41621574-113-85-90
    Average 99 stars, based on 48653 article reviews
    transmission 268 electron microscope - by Bioz Stars, 2026-09
    99/100 stars

    Images

    Related Articles

    Control:

    Article Title: DPhA-EtOBz-TSC Targets Cystathionine γ-Lyase (CSE) to Trigger Ferroptosis and Inhibit Colorectal Cancer Growth In Vitro and In Vivo.
    Article Snippet: 34 Colorectal cancer (CRC) remains one of the most prevalent malignancies globally, 35 with limited therapeutic options for advanced-stage patients due to acquired drug 36 resistance and systemic toxicity.. Ferroptosis, an iron-dependent form of regulated 37 cell death driven by lipid peroxidation, has emerged as a promising therapeutic 38 target for CRC, while cystathionine γ-lyase (CSE)—a key enzyme in hydrogen sulfide 39 (H2S) biosynthesis—plays a critical role in maintaining redox homeostasis and 40 suppressing ferroptosis in cancer cells.. Here, we synthesized a novel small-molecule 41 compound, DPhA-EtOBz-TSC, and systematically evaluated its anti-CRC efficacy and 42 underlying molecular mechanism.

    Staining:

    Article Title: DPhA-EtOBz-TSC Targets Cystathionine γ-Lyase (CSE) to Trigger Ferroptosis and Inhibit Colorectal Cancer Growth In Vitro and In Vivo.
    Article Snippet: 34 Colorectal cancer (CRC) remains one of the most prevalent malignancies globally, 35 with limited therapeutic options for advanced-stage patients due to acquired drug 36 resistance and systemic toxicity.. Ferroptosis, an iron-dependent form of regulated 37 cell death driven by lipid peroxidation, has emerged as a promising therapeutic 38 target for CRC, while cystathionine γ-lyase (CSE)—a key enzyme in hydrogen sulfide 39 (H2S) biosynthesis—plays a critical role in maintaining redox homeostasis and 40 suppressing ferroptosis in cancer cells.. Here, we synthesized a novel small-molecule 41 compound, DPhA-EtOBz-TSC, and systematically evaluated its anti-CRC efficacy and 42 underlying molecular mechanism.

    Transmission Assay:

    Article Title: DPhA-EtOBz-TSC Targets Cystathionine γ-Lyase (CSE) to Trigger Ferroptosis and Inhibit Colorectal Cancer Growth In Vitro and In Vivo.
    Article Snippet: 34 Colorectal cancer (CRC) remains one of the most prevalent malignancies globally, 35 with limited therapeutic options for advanced-stage patients due to acquired drug 36 resistance and systemic toxicity.. Ferroptosis, an iron-dependent form of regulated 37 cell death driven by lipid peroxidation, has emerged as a promising therapeutic 38 target for CRC, while cystathionine γ-lyase (CSE)—a key enzyme in hydrogen sulfide 39 (H2S) biosynthesis—plays a critical role in maintaining redox homeostasis and 40 suppressing ferroptosis in cancer cells.. Here, we synthesized a novel small-molecule 41 compound, DPhA-EtOBz-TSC, and systematically evaluated its anti-CRC efficacy and 42 underlying molecular mechanism.

    Microscopy:

    Article Title: DPhA-EtOBz-TSC Targets Cystathionine γ-Lyase (CSE) to Trigger Ferroptosis and Inhibit Colorectal Cancer Growth In Vitro and In Vivo.
    Article Snippet: 34 Colorectal cancer (CRC) remains one of the most prevalent malignancies globally, 35 with limited therapeutic options for advanced-stage patients due to acquired drug 36 resistance and systemic toxicity.. Ferroptosis, an iron-dependent form of regulated 37 cell death driven by lipid peroxidation, has emerged as a promising therapeutic 38 target for CRC, while cystathionine γ-lyase (CSE)—a key enzyme in hydrogen sulfide 39 (H2S) biosynthesis—plays a critical role in maintaining redox homeostasis and 40 suppressing ferroptosis in cancer cells.. Here, we synthesized a novel small-molecule 41 compound, DPhA-EtOBz-TSC, and systematically evaluated its anti-CRC efficacy and 42 underlying molecular mechanism.



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